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Hemolytic iron regulation in traumatic brain injury and alcohol use
Agnieszka Agas
, Arun Reddy Ravula
, Xiaotang Ma
, Yiming Cheng
,
Kevin D. Belfield
, James Haorah
College of Science and Liberal Arts
Office of the Dean - CSLA
Chemistry and Environmental Science
Bio-Medical Engineering
Research output
:
Contribution to journal
›
Article
›
peer-review
1
Scopus citations
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Keyphrases
Traumatic Brain Injury
100%
Alcohol Use
100%
Iron Regulation
100%
Alcohol
22%
Red Blood Cells
22%
Alcohol Consumption
22%
Iron Management
22%
Central Nervous System
11%
Energy Release
11%
Major Components
11%
Management System
11%
Chronic Alcohol Consumption
11%
Induced Activation
11%
Energy Depletion
11%
Microglia
11%
Ferritin
11%
Chronic Alcohol
11%
Induced Expression
11%
Neurotoxicity
11%
Rat Model
11%
Expression Profile
11%
Regulatory Proteins
11%
Microglial Activation
11%
Heme oxygenase-1 (HO-1)
11%
Free Iron
11%
Fluid Percussion
11%
Lipocalin 2 (LCN2)
11%
Ferritin Light Chain
11%
Alcohol Feeding
11%
Ferroptosis
11%
Cell Clearance
11%
Biochemistry, Genetics and Molecular Biology
Hemocyte
100%
Microglia
100%
Liquid
50%
Heme Oxygenase
50%
Ferritin
50%
Lipocalin-2
50%
Ferritin Light Chain
50%
Rat Model
50%
Ferroptosis
50%
Neuroscience
Neurotrauma
100%
Traumatic Brain Injury
100%
Alcohol Abuse
100%
Microglia
22%
Hemocyte
22%
Ferritin
22%
Central Nervous System
11%
Neurotoxicity
11%
Light Chain
11%
Feeding Behavior
11%
Regulator Protein
11%
Ferroptosis
11%
Heme Oxygenase 1
11%
Neutrophil Gelatinase Associated Lipocalin
11%