Modeling the neuroprotective role of enhanced astrocyte mitochondrial metabolism during stroke

Keyphrases

• Astrocytes 100%
• GTP-binding Proteins 100%
• Mitochondrial ATP Production 100%
• Neuroprotective Role 100%
• Cerebral Ischemic Stroke 100%
• Mitochondrial Metabolism 100%
• Protein-coupled Receptor 100%
• Mathematical Model 50%
• Protective Effect 50%
• Model Support 50%
• Biological Data 50%
• Varying Condition 50%
• Astrocytic 50%
• Rate Limiting 50%
• Endoplasmic Reticulum Ca2+ 50%
• Energy Substrates 50%
• Cell Volume 50%
• Explicit Formula 50%
• Ca2+ Handling 50%
• Ca2+ Release 50%
• Ion Homeostasis 50%
• Cell Membrane Potential 50%
• Receptor Signaling 50%
• P2Y1R 50%

Neuroscience

• Neuroprotective Agent 100%
• Astrocyte 100%
• Eicosanoid Receptor 100%
• G Protein 100%
• Brain Ischemia 100%
• Mitochondrial Respiration 100%
• Adenosine Triphosphate 100%
• Behavior (Neuroscience) 50%
• Cell Membrane Potential 50%
• Endoplasmic reticulum 50%
• Ion Homeostasis 50%

Biochemistry, Genetics and Molecular Biology

• Eicosanoid Receptor 100%
• Astrocyte 100%
• GTP Binding Protein 100%
• Mitochondrial Respiration 100%
• Adenosine Triphosphate 100%
• Dynamics 50%
• Homeostasis 50%
• Cell Membrane Potential 50%
• Cell Volume 50%